Alzheimers News /alzheimersnews Alzheimers News - Alzheimers information Tue, 04 Apr 2017 19:19:03 +0000 en-US hourly 1 Tea found to play a major role in the fight against dementia /alzheimersnews/2017-04-02-tea-found-to-play-a-major-role-in-the-fight-against-dementia.html Mon, 03 Apr 2017 04:20:55 +0000 Cut your dementia risk in half by simply drinking a cup or two of tea every day. Scientists have found that the refreshing drink is not only good for your tummy but for your brain too. These benefits can be seen regardless of the variety of tea you prefer, be it green, black, or oolong. Moreover, those that are predisposed to this cognitive illness — the “dementia gene” as it were — can significantly reduce their risk of the disease by around 80 percent.

A new study, released by the National University of Singapore (NUS), concluded that the polyphenols found in the tea leaves promote cognitive health. Also, other compounds in tea leaves, such as catechins and theaflavins, have anti-inflammatory and antioxidant properties that decrease the potential for brain decline.

Dr. Feng Lei, who led the study, said: “Because tea is cheap, non-toxic and widely consumed, it has huge potential in promoting cognitive health and perhaps delaying the onset of dementia.”

Tea Time (for your brain)

Tea has been a recommended drink for anxiety and stress relief. The various scents and flavors trigger certain parts of the brain associated with relaxation. Popular media encourages this further with pictures of people smiling contently — or perhaps discussing a point in philosophy — over a steaming cup of tea. Tea has also become synonymous with wisdom.

Yet for all its assumed brain-enhancing properties, there has been limited research studying tea’s actual physiological effects. The study by the NUS provides undeniable proof that diet directly impacts mental health.

Researchers studied the lifestyle habits of 2,500 people who were aged 55 and over and looked at how much tea they drank. Participants were tested on their cognitive function. Initial conclusions validated their hypothesis: Those that drank the most tea performed better compared to those who did not. When the experiment was repeated two years later, the results were even more astounding. Those that drank the most black tea showed the least cognitive decline. They found that other factors — such as eating habits — come into play as well but there is a general consensus that drinking tea regularly keeps the brain working properly.

People who drank two to three cups daily reduced their risk of developing dementia by 55 percent. Meanwhile, taking six to ten cups a day can reduce the risk by 63 percent. These findings were published in the American Journal of Clinical Nutrition.

The mechanics are believed to work as such: Polyphenols which are abundant in tea prevent the oxidation of brain cells. Typically, aging brains show the most oxidation. This progressive decline of function makes the elderly more susceptible to brain disease. By preventing the oxidation, tea keeps brains young.  Secondly, polyphenols are believed to prevent the buildup of plaque in the brain. Similar to cardiac problems caused by unwanted deposits, neural connections can become blocked. These pathways are necessary for memory and attention. Inhibited brain function can lead to dementia and Alzheimer’s. (RELATED: Find more news about preventing brain disorders at

Tea vs. Coffee: The Battle of the Stimulants

Once, tea was lauded only for its diuretic and stimulant properties. Numerous studies have added more reasons to add this drink into one’s diet. It has been proven that tea can lower cholesterol levels, reduce the risk of certain cancers, and even reduce blood clots in the body. With this new study, tea has taken a higher status.

Dr. Lei though is quick to mention that coffee does have its own benefits. A 2009 study found that drinking black, unsweetened coffee in midlife can reduce cognitive decline. More research is necessary to determine both drinks’ capacities for maintaining optimum cognitive function.

“Despite high-quality drug trials, effective pharmacological therapy for neurocognitive disorders such as dementia remains elusive and current prevention strategies are far from satisfactory,” he said.






Aluminum linked directly to early onset of Alzheimer’s disease /alzheimersnews/2017-02-17-aluminum-linked-directly-to-early-onset-of-alzheimers-disease.html Fri, 17 Feb 2017 17:30:44 +0000 Aluminum is everywhere: it contaminates vaccines, it’s in a variety of medications, baby products, cosmetics, and it’s even in the food you eat. And like several other metals, it isn’t really all that great for the human brain (or the rest of the body).

Even the CDC’s Agency for Toxic Substances and Disease Registry (ATSDR) notes that aluminum can elicit negative effects in the musculoskeletal, neurological, and respiratory systems. This is especially worrisome because some research indicates that aluminum is capable of building up in bodily tissues, which would greatly increase its potential to cause harm. [RELATED: Keep up with the latest CDC headlines at]

Research has suggested that there may be a link between aluminum exposure and Alzheimer’s disease for several years. That link, however, has always been somewhat murky. The evidence to support their claims has often been lacking. Recently, however, scientists have found a direct link between the metal and the onset of the neurological disorder.

Connecting the dots: aluminum and Alzheimer’s

According to scientists from Keele University, located in Staffordshire, aluminum actually plays a role in most — if not all– cases of Alzheimer’s. Professor Exley, a scientist from the university, has been studying this connection at length. In a recent article for The Hippocratic Post, Exley explained, “We already know that the aluminium content of brain tissue in late-onset or sporadic Alzheimer’s disease is significantly higher than is found in age-matched controls. So, individuals who develop Alzheimer’s disease in their late sixties and older also accumulate more aluminium in their brain tissue than individuals of the same age without the disease.”

Exley went on to say that even higher levels of aluminum have been found in individuals with certain forms of Alzheimer’s disease and notes that these high amounts of exposure are often attributed to the environment these people live in, or their workplace. “This means that Alzheimer’s disease has a much earlier age of onset, for example, fifties or early sixties, in individuals who have been exposed to unusually high levels of aluminium in their everyday lives,” contends Exley.

High aluminum content in Alzheimer’s patients

In 2016, Exley published his most revealing study yet in the Journal of Trace Elements in Medicine and Biology. This study is believed to be of exceptional value because it is the first to measure aluminum content in the brain tissue of individuals that have been diagnosed with familial Alzheimer’s disease.

Alzheimer’s is considered to be “familial” when two or more people in the same family are stricken by the condition.

Exley and his team found that people who had passed away with diagnosed familial Alzheimer’s disease had the highest concentrations of aluminum in their brain tissue that had ever been recorded.

“We now show that some of the highest levels of aluminium ever measured in human brain tissue are found in individuals who have died with a diagnosis of familial Alzheimer’s disease,” Exley wrote. He went on to note that the amount of aluminum found in the brain tissue of the individuals with familial Alzheimer’s disease were almost identical to those seen in individuals who died of aluminum-induced encephalopathy while undergoing renal dialysis. [RELATED: Learn more about toxic metals and other damaging compounds at]

Exley and his team concluded that their research indicates that the genetic predisposition for Alzheimer’s disease is likely tied to the accumulation of aluminum in brain tissue. The researchers note that aging is a risk factor for Alzheimer’s and that the human brain tends to accumulate more aluminum as we get older. Because of aluminum’s neurotoxic effects, its accumulation in brain is going to exacerbate or contribute to any ongoing disease or toxicity.


Odds of early death in Alzheimer’s patients nearly doubles after taking antipsychotic drugs /alzheimersnews/2017-01-04-odds-of-early-death-in-alzheimers-patients-nearly-doubles-after-taking-antipsychotic-drugs.html Thu, 05 Jan 2017 04:50:25 +0000 Alzheimer’s patients who take antipsychotic drugs have a significantly increased risk of dying early compared to those who don’t, new research out of Finland has found. An in-depth analysis of nearly 58,000 people diagnosed with Alzheimer’s disease between 2005 and 2011 found that those who take popular antipsychotics like Abilify (aripiprazole) or Risperdeal (risperidone) face as much as a 60 percent increased risk of premature death — and this risk is even higher when Alzheimer’s patients take two or more antipsychotics.

Published in the Journal of Alzheimer’s Disease, the paper found that premature death risk is highest when Alzheimer’s patients first start taking an antipsychotic drug, though long-term use poses similar health risks. The worst scenario is Alzheimer’s patients who take two or more antipsychotics, as their risk of premature death is upwards of twice the normal rate, a shocking figure that backs earlier research into the adverse effects associated with these common pharmaceutical drugs. Perhaps more natural healing methods should be analyzed as well.

Marjaana Koponen, a doctoral student from the School of Pharmacy at the University of Eastern Finland and lead author of the new study, stated that even though the research can’t officially prove a cause-and-effect link between antipsychotics and early death, they are highly suggestive of this. Some of the earliest research on the subject from roughly 10 years ago arrived at similar conclusions, mainly that antipsychotics are high-risk and need to be evaluated more closely when it comes to their use in people with dementia.

One such study from more recently, published in the journal JAMA Psychiatry in 2015, found that antipsychotic drugs marketed as treating hallucinations, delusions, agitation, and aggression are directly associated with higher rates of premature death. Of specific concern is an “atypical,” mood-stabilizing antipsychotic drug known as Depakene (valproic acid), which researchers from the University of Michigan Medical School in Ann Arbor say shouldn’t even be prescribed to dementia patients.

“The harms associated with using these drugs in dementia patients are clear, yet clinicians continue to use them,” Dr. Donovan T. Maust, M.D., one of the lead authors of this earlier study that looked at more than 90,000 U.S. veterans with dementia, told Neurology Advisor last year. “That’s likely because the symptoms are so distressing. These results should raise the threshold for prescribing further.”

Research going back years shows extreme dangers with antipsychotic use

Going back even further, a 2012 paper published in the American Journal of Psychiatry raised concerns about the use of antipsychotic drugs in Alzheimer’s patients, noting that other non-drug interventions must be thoroughly exhausted before even considering the use of mind-altering medications in this sensitive segment of the population. Besides the serious cardiovascular effects, antipsychotics seem to significantly increase patients’ risk of pneumonia, and many of those who develop this disease end up dying.

Each of these studies reiterates the dangers associated with antipsychotic drugs, especially in people suffering from some form of dementia. Antipsychotics should only be used, most researcher generally agree, in extreme cases where dementia symptoms are well beyond the scope of what can be reasonably managed using other means. And even then, such drugs need to be used in very small quantities for a very short amount of time, and under the careful supervision of a qualified physician.

“From my perspective, the correct management behavioural problems in dementia is nearly always reducing medications, not starting them,” says Professor David Le Couteur from the Australasian Society of Clinical and Experimental Pharmacologists and Toxicologists, about the use of antipsychotics.

“People with dementia are human beings and they need to be treated with respect and sedating them because of their behaviour just feels wrong as a human being.”


Aluminum in vaccines linked to Alzheimer’s and other neurological conditions /alzheimersnews/2016-12-29-aluminum-in-vaccines-linked-to-alzheimers-and-other-neurological-deterioration.html Fri, 30 Dec 2016 02:46:30 +0000 British researchers claim they have confirmed that aluminum plays a strong role in the onset of Alzheimer’s disease.

Dementia and Alzheimer’s disease are now the leading causes of death around the world, superseding heart disease. More than 5 million persons in the U.S., where it is the sixth leading cause of death, have been diagnosed with Alzheimer’s, according to the Alzheimer’s Association; in the U.K., 850,000 people are living with the brain disorder.

By mid century, the number of Alzheimer’s suffers could range from 14 to 16 million.

Aluminum is said to be the most widely used metal on the planet and is found in cookware, aspirin, antacids, baking soda, and flour, as well as vaccines. And let’s not forget the old reliable aluminum foil.

Natural News previously reported that in a 15-year study of French elderly men and women, regular consumption of tap water was associated with aluminum toxicity and increased prevalence of dementia.

Many childhood vaccines also contain aluminum, as Natural News has separately detailed. Aluminum is included in vaccines as an “adjuvant,” a component that boosts the body’s short-term immune response in order to produce antibodies to the vaccine agent faster. This very function may be part of what makes aluminum in vaccines risky. Aluminum also is a neurotoxin that has reportedly been linked to various types of brain damage in both kids and adults.

A 2013 study in the journal Immunologic Research apparently confirmed that aluminum toxicity has a negative impact on the body’s nervous system “across the age span.” In adults, over-exposure to aluminum in the system can lead to age-related neurological conditions that resemble Alzheimer’s disease. Similar outcomes were observed in laboratory animals, the researchers noted.

In the new study, Professor Christopher Exley of Keele University, one of the authors, explains that a study published in the Journal of Trace Elements in Medicine and Biology makes the link between human exposure to aluminum (or aluminium as the metal is known in Britain) and Alzheimer’s disease “ever more compelling” and overwhelming than originally thought.

While aluminum is not the only factor in dementia, he noted, it is an important one, and he called upon those who reject the aluminum-Alzheimer’s link to reevaluate their position based on the new evidence.

Writing in The Hippocratic Post blog, Exley outlined what he and his colleagues discovered when they examined brain matter from 12 deceased Alzheimer’s sufferers.

“In my view, the findings are unequivocal in their confirmation of a role for aluminium in some if not all Alzheimer’s disease…We now show that some of the highest levels of aluminium ever measured in human brain tissue are found in individuals who have died with a diagnosis of familial Alzheimer’s disease…This new research may suggest that these genetic predispositions to early onset Alzheimer’s disease are linked in some way to the accumulation of aluminum (through ‘normal’ everyday human exposure) in brain tissue.”

Summing up what Exley and his co-authors determined, the study explains that “Aluminium is neurotoxic and and the concentrations of aluminium found in these familial [Alzheimer’s disease] brains are unlikely to be benign and indeed are highly likely to have contributed to both the onset and the aggressive nature of any ongoing AD in these individuals. These data lend support to the recent conclusion that brain aluminium will contribute towards all forms of [Alzheimer’s disease] under certain conditions.”

As far as preventive measures are concerned, Prof. Exley’s blog concludes with a call to action that “We should take all possible precautions to reduce the accumulation of aluminium in our brain tissue through our everyday activities and we should start to do this as early in our lives as possible.





“Overwhelming” evidence that aluminum causes Alzheimer’s disease… shocking new science removes all doubt /alzheimersnews/2016-12-27-overwhelming-evidence-that-aluminum-causes-alzheimers-disease-shocking-new-science-removes-all-doubt.html Tue, 27 Dec 2016 17:51:26 +0000 Scientists have long known that there is a link between aluminum and Alzheimer’s disease, but many have said that there was not enough actual evidence to prove a relationship between the debilitating illness and a common metal used by scores of people every day.

But now, Prof. Chris Exley, of Keele University in England, states unequivocally that his latest research confirms that aluminum definitely plays a role in the development of the mentally debilitating disease, the UK’s Daily Mail reported.

In an article for the medical blogging website The Hippocratic Post, Exley laid out the details of his latest research.

Exley writes that the study, completed by his group and published in the Journal of Trace Elements in Medicine and Biology, “makes the link even more compelling.” He says that in his view, the link is distinct and a compelling confirmation that aluminum plays a role in some, if not all, cases of Alzheimer’s disease.

“At the very least,” he writes, “these new results should encourage everyone and even those who have steadfastly maintained that aluminum has no role in the disease to think again.”

While the researcher said he does not believe that aluminum is the only factor in the development of the disease, he says it most definitely is an important factor and ought to be considered as such.

Exley said when the results of his new study were added to what was already known about the link, their significance “becomes overwhelming and compelling.”

The more aluminum exposure at an earlier age, the more likely you will develop Alzheimer’s

He said scientists were already aware that the aluminum content of brain tissue in late-onset or sporadic Alzheimer’s disease is much higher than content levels found in control cases. That means people who develop Alzheimer’s in their late sixties and beyond also are accumulating more aluminum in the brain than people who are the same age but devoid of the disease.

What scientists have also discovered previously is that people who have developed familial Alzheimer’s also produce more amyloid beta than non-Alzheimer’s individuals, and the onset of the disease comes much sooner in life.

The new research further suggests, then, that those genetic predispositions to early onset Alzheimer’s are likely linked in some way “to the accumulation of aluminum (through ‘normal’ everyday human exposure) in brain tissue,” he wrote.

What’s more, scientists have also known that aluminum is a neurotoxin, meaning its accumulation in the brain at any age can lead to toxicity and a wide range of additional health problems.

Exley also said that environmental or occupational exposure to aluminum over the course of time will result in higher levels of aluminum accumulating in brain tissue, and thus, an earlier onset of Alzheimer’s disease.


Light as medicine: Flashing lights found to halt Alzheimer’s disease /alzheimersnews/2016-12-16-light-as-medicine-flashing-lights-found-to-halt-alzheimers-disease.html Fri, 16 Dec 2016 17:24:55 +0000 (NaturalNews) Lights flashing at a precise frequency might hold promise as a treatment for Alzheimer’s disease, according to a study conducted by researchers from the Massachusetts Institute of Technology, and published in the journal Nature.

Significantly, the treatment worked by activating the brain’s own dedicated immune system, causing the brain to clear out the amyloid plaques that are the physiological signature of the disease.

Alzheimer’s is the most common form of dementia, which is characterized by progressive decline in memory and cognitive function. No form of dementia has any known cure or effective treatment.

Dementia is now the top killer in the United Kingdom. In the United States, it ranks third behind heart disease and cancer. Worldwide, rates of dementia continue to rise.

Striking, but short-lived

Mice with amyloid plaques in their brains were exposed to LEDs flickering at a frequency of 40 Hz, the same frequency as the brain waves known as gamma oscillations. They found that in the visual cortex – one of the first areas to develop amyloid plaques in mice – levels of gamma oscillations immediately increased, accompanied by a dramatic increase in the activity of immune cells known as microglia, which play a key role in clearing debris from the brain.

The flicker is soft enough that it can barely be noticed by the human eye.

Within an hour, there were noticeable decreases in the amyloid concentrations in the treated mice’s visual cortexes. Overall, the concentration of amyloids decreased about 50 percent following treatment.

Within about a day, however, amyloid plaques had returned to their prior levels. So the researchers gave mice daily treatments with the LED box for a week. At the end of the week, they found that treated mice had amyloid levels 67 percent lower than untreated mice, and that their plaques were an average of 64 percent smaller.

Previously, the researchers had conducted a study that consisted of implanting LEDs into the brains of animals to see if an oscillating light could induce gamma oscillations. They focused on these oscillations because they are a type of brain wave that is significantly reduced in areas of the brain affected by Alzheimer’s. That study found that not only did the lights induce gamma oscillations, but they led to an increase in microglia activity.

The treatment was considered too invasive for human patients, however, so the researchers followed up with the new study.

Major hurdles remain

The major problem with the recent research is that the benefits of the light do not appear to have extended much beyond the visual cortex of the mice. But the human visual cortex does not tend to accumulate amyloid plaques; instead, these plaques concentrate in regions such as the hippocampus, which are believed to regulate cognition and memory.

Nevertheless, the researchers have formed a company, Cognito Therapeutics, that has already petitioned the FDA to begin trials in human Alzheimer’s patients.

“So many things have been shown to work in mice, only to fail in humans,” lead researcher Dr. Li Huei Tsai said. “But if humans behave similarly to mice in response to this treatment, I would say the potential is just enormous.”

If effective, the treatment may also have potential for other neuropsychiatric conditions characterized by reduced gamma oscillations, including autism, Parkinson’s disease and schizophrenia.

To date, no drugs targeting amyloid plaques have shown effectiveness in treating Alzheimer’s disease. But the researchers say that those interventions had either not acted early enough, or had not found an ideal method of affecting the plaque buildup.

“I think we have something very fundamentally different,” Tsai said.

Tsai noted that if it can be developed into a treatment, light therapy has the advantage of not needing to bypass the blood-brain barrier, as drugs do. As a non-pharmaceutical treatment, it is likely to have significantly fewer side effects.

Most significantly, Tsai emphasized that the treatment relies on the body’s own immune system, rather than an external agent.

“We just directly recruit other neurons and other cell types in the brain to sort of enable the brain’s inner ability to repair itself,” Tsai said.

Sources for this article include:

Avoid Alzheimer’s disease with these 5 diet tips /alzheimersnews/2016-09-23-avoid-alzheimers-disease-with-these-6-diet-tips.html Fri, 23 Sep 2016 20:19:19 +0000 Alzheimer’s disease is a devastating illness that affects over 5 million Americans alone. It is also the sixth leading cause of death in America. The Alzheimer’s Association estimates that roughly 1-in-3 senior citizens will die with Alzheimer’s disease, or another form of dementia. It kills more people than breast and prostate cancer combined. While there are many things about Alzheimer’s disease that have yet to be understood, there are ways you can help protect yourself against the disease. While it is largely considered to be an age-related condition with ties to genetic predisposition, nothing is ever absolute — especially when it comes to human health. These five dietary staples can help boost brain health and may even prevent dementia:

1. Ginger, berries and olive oil

What do these foods have in common? They can help protect your brain’s glial cells, which are imperative for the health of your central and peripheral nervous systems. Glial cells help moderate neurological functions, remove damaged cells, and help repair damage. Research has shown that when glial cells fail to do their jobs, it leads to severe damage in neurons (another type of brain cell), which quickly leads to cognitive impairments.

2. Green Tea

Studies show the polyphenols in green tea can actually provide benefits to adults with cognitive deficits. They may also lend neuroprotective benefits and may also help stave off further impairments in dementia patients.

3. Keep Your Blood Sugar Stable

A link between Type 2 Diabetes and Alzheimer’s disease has yet to be discerned. But, there is substantial evidence that high blood sugar negatively effects the brain and can cause lasting damage. Eating smaller meals and avoiding junk foods that contain refined grains and added sugars can help to keep your blood sugar on an even keel.

4. Kale, Spinach and Asparagus

Studies indicate that eating at least one full serving of leafy greens each day can help keep your brain young. Their high vitamin K concentration can also help prevent deficiency, which is linked to accelerating Alzheimer’s development.

5. Fatty Fish

DHA and EPA are two omega-3 fatty acids that are known to promote brain health, and prevent dementia. Because these essential nutrients cannot be produced by the body, it is very important to get enough through diet. Wild-caught fatty fishes such as salmon, trout and sardines are great choices for getting enough omega-3 fatty acids. A supplement from a reputable manufacturer is also a great option.


Why do some researchers refer to Alzheimer’s as the ‘type-3’ diabetes? /alzheimersnews/2016-07-15-why-do-some-researchers-refer-to-alzheimers-as-the-type-3-diabetes.html Fri, 15 Jul 2016 14:57:15 +0000 Alzheimer’s disease is a terrifying brain dementia that tears families and loved ones apart. But what if the disease isn’t Alzheimer’s? What if the dementia is really a form of diabetes? And what if a proper organic whole food diet can make the difference? This explosive discovery was made by Dr. Suzanne de la Monte from Rhode Island Hospital.

Natural News reports, “[Dr. de la Monte discovered] “that diabetes is closely associated with several key neuronal factors implicated in dementia. . . Alzheimer’s progresses as a result of the brain developing resistance to insulin, which in turn prevents proper lipid (fat) metabolism. Over time, these lipids build up in the brain rather than properly absorb, which results in increased stress and inflammation, as well as the symptoms commonly associated with dementia.” shares more insights on Dr. de la Monte’s work:

“Insulin in the body is not only produced in the pancreas, as commonly known, but in the brain. De la Monte found that in Alzheimer’s disease, the production of insulin and similar substances in the brain almost cease, creating what she calls ‘diabetes in the brain . . .’

Dr. de la Monte believes that processed, industrial food is a key factor. reports that “fast food and meat meat processing increased eightfold from 1970 to 2005. . . Grain consumption increased fivefold. Rates of Alzheimer’s, diabetes, and fatty liver disease increased dramatically in people ages 55 and over during this period. . .”

Eliminate statins and change your diet, reports Natural News.

“Consuming more healthy saturated fats like coconut oil can . . . help repair the inflammation problem that promotes the progression of Alzheimer’s, [and] increases the absorption of cholesterol in the brain. . . [With] Type-2 and Type-3 diabetes  . . . There has been tremendous success in controlling and eliminating diabetes through a low-carb and high-fat diet.”



Scientists prove THC can eradicate toxic proteins associated with Alzheimer’s /alzheimersnews/2016-06-30-scientists-prove-thc-can-eradicate-toxic-proteins-associated-with-alzheimers.html Thu, 30 Jun 2016 15:11:06 +0000 Alzheimer’s disease is one of the most terrifying and debilitating illnesses imaginable, so combating it is especially important.

Scientists at Salk Institute have found new evidence that supports the theory that tetrahydrocannabinol (THC) and many other compounds found in cannabis can help with the prevention of Alzheimer’s disease.

Science Daily reports, “The researchers found that high levels of amyloid beta were associated with cellular inflammation and higher rates of neuron death. They demonstrated that exposing the cells to THC reduced amyloid beta protein levels and eliminated the inflammatory response from the nerve cells caused by the protein, thereby allowing the nerve cells to survive.”

Essentially, they’ve found that cannabis is a safe, effective way to prevent Alzheimer’s disease from setting in. The evidence is there and everyone at risk should definitely consider trying out THC. Since we know that marijuana isn’t the dangerous, life-ending drug that the mainstream media has made it out to be, what is there to lose?

Once again, the health benefits of cannabis have been revealed in a way that should completely eradicate any belief that the plant is a “harmful” or “damaging” substance. We’re long past the point where that should even be up for debate.

Alzheimer’s is such an awful thing to experience that the federal government should be doing everything in their power to prevent the American people from suffering from it. If that includes legalizing cannabis, then it’s time to kick-start the inevitable.

This is absolutely groundbreaking information that should immediately translate to marijuana’s legalization — yet there’s little doubt that the federal government will continue to keep cannabis behind bars for their own financial benefit. We’re onto their scheme and it’s time for the United States citizens to loudly support legalization. With enough support, the powers that be will no longer be able to ignore the voices of the American people.

With enough support, we may finally have legal access to a plant with numerous healing properties.



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